Cytotoxicity of activator expression in CRISPR-based transcriptional activation systems

CRISPR-based transcriptional activation (CRISPRa) has extensive research and clinical potential. Here, we show that commonly used CRISPRa systems can exhibit pronounced cytotoxicity. We demonstrate the toxicity of CRISPRa vectors expressing the activation domains (ADs) of the transcription factors p65 and HSF1, components of the synergistic activation mediator (SAM) CRISPRa system. Based on our findings for the SAM system, we extended our studies to additional ADs and acetyltransferase core domains. We show that the expression of potent transcriptional activators in lentiviral producer cells can lead to low lentiviral titers, while their expression in the transduced target cells leads to cell death. Using inducible lentiviral vectors, we could not identify an activator expression window for effective SAM-based CRISPRa without measurable toxicity. The toxicity of current SAM-based CRISPRa systems hinders their wide adoption in biomedical research and introduces selection pressures that may confound genetic screens. Our results suggest that the further development of CRISPRa technology should consider both the efficiency of gene activation and activator toxicity.

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Publication Details

Journal
Nature Communications
Published
2025-08-29
DOI
https://doi.org/10.1038/s41467-025-63570-4
Citations
3
Primary Topic
CRISPR and Genetic Engineering
Type
article
Field-Weighted Citation Impact
2.42

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article

Cytotoxicity of activator expression in CRISPR-based transcriptional activation systems

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3 citations
Nature Communications
CRISPR and Genetic Engineering
2.42
article

Cytotoxicity of activator expression in CRISPR-based transcriptional activation systems

Elizabeth T. Bartom, Ajinkya Patil, Subrata Roy, Eva Gottwein, Jesus A. Ortega, Mark Manzano, Josh Tycko, Christine B. Magdongon, Ziyan Liang, Shreya Jambardi, Aakaanksha Maddineni
article en
3 citations

Abstract

CRISPR-based transcriptional activation (CRISPRa) has extensive research and clinical potential. Here, we show that commonly used CRISPRa systems can exhibit pronounced cytotoxicity. We demonstrate the toxicity of CRISPRa vectors expressing the activation domains (ADs) of the transcription factors p65 and HSF1, components of the synergistic activation mediator (SAM) CRISPRa system. Based on our findings for the SAM system, we extended our studies to additional ADs and acetyltransferase core domains. We show that the expression of potent transcriptional activators in lentiviral producer cells can lead to low lentiviral titers, while their expression in the transduced target cells leads to cell death. Using inducible lentiviral vectors, we could not identify an activator expression window for effective SAM-based CRISPRa without measurable toxicity. The toxicity of current SAM-based CRISPRa systems hinders their wide adoption in biomedical research and introduces selection pressures that may confound genetic screens. Our results suggest that the further development of CRISPRa technology should consider both the efficiency of gene activation and activator toxicity.

Nature CommunicationsVol. 16(1)
Northwestern University (US), Harvard University (US), University of Arkansas for Medical Sciences (US)
Northwestern University, National Institutes of Health, National Cancer Institute, National Institute of Allergy and Infectious Diseases
Zero hunger
Openalex Percentile: Top 10%
CRISPR and Genetic Engineering
2.42
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