MiR-193-3p suppresses phenotypic switching in vascular smooth muscle cells by regulating IGF2 during in-stent restenosis

These findings suggest the miR-193-3p may serve as a potential biomarker for ISR and that targeting the miR-193-3p/IGF2 axis could be a promising strategy for managing ISR.

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Publication Details

Journal
American Journal of Translational Research
Published
2025-01-01
DOI
https://doi.org/10.62347/nzrn6858
Primary Topic
Cancer-related molecular mechanisms research
Type
article
Field-Weighted Citation Impact
0.00

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article

MiR-193-3p suppresses phenotypic switching in vascular smooth muscle cells by regulating IGF2 during in-stent restenosis

Tao Lv
American Journal of Translational Research
Cancer-related molecular mechanisms research
article

MiR-193-3p suppresses phenotypic switching in vascular smooth muscle cells by regulating IGF2 during in-stent restenosis

Tao Lv
article en

Abstract

These findings suggest the miR-193-3p may serve as a potential biomarker for ISR and that targeting the miR-193-3p/IGF2 axis could be a promising strategy for managing ISR.

American Journal of Translational ResearchVol. 17(8)
Northwest University (CN)
Xi'an Municipal Bureau of Science and Technology
Openalex Percentile: Top 72%
Cancer-related molecular mechanisms research
DOIOpen Access PDFOpenAlex
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MiR-193-3p suppresses phenotypic switching in vascular smooth muscle cells by regulating IGF2 during in-stent restenosis — Tao Lv · American Journal of Translational Research (2025) | TGRS Research Map | TGRS