Glycyrrhizic Acid Ameliorates <scp>LPS</scp>‐Induced <scp>WI</scp>‐38 Cell Inflammation, Oxidative Stress, and Ferroptosis via Targeting <scp>METTL14</scp> in Infantile Pneumonia

GA alleviates LPS-induced WI-38 cell cytotoxicity, inflammation, oxidative stress and ferroptosis by METTL14 knockdown. Our findings suggest that GA may pave the way for the treatment of infantile pneumonia.

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Publication Details

Journal
Clinical and Experimental Pharmacology and Physiology
Published
2025-08-27
DOI
https://doi.org/10.1111/1440-1681.70068
Citations
1
Primary Topic
Cancer-related molecular mechanisms research
Type
article
Field-Weighted Citation Impact
0.94
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article

Glycyrrhizic Acid Ameliorates LPS‐Induced WI‐38 Cell Inflammation, Oxidative Stress, and Ferroptosis via Targeting METTL14 in Infantile Pneumonia

Xiaofei Xie, Caiwen Wang, Zhuang Wang, Ting Sun et al.
1 citations
Clinical and Experimental Pharmacology and Physiology
Cancer-related molecular mechanisms research
0.94
article

Glycyrrhizic Acid Ameliorates LPS‐Induced WI‐38 Cell Inflammation, Oxidative Stress, and Ferroptosis via Targeting METTL14 in Infantile Pneumonia

Xiaofei Xie, Caiwen Wang, Zhuang Wang, Ting Sun, Na Wang, Yingying Sun, Yongji Wang, Yongfu Song
article en
1 citations

Abstract

GA alleviates LPS-induced WI-38 cell cytotoxicity, inflammation, oxidative stress and ferroptosis by METTL14 knockdown. Our findings suggest that GA may pave the way for the treatment of infantile pneumonia.

Clinical and Experimental Pharmacology and PhysiologyVol. 52(10)
Changchun University of Chinese Medicine (CN), The Affiliated Hospital to Changchun University of Chinese Medicine (CN)
Openalex Percentile: Top 21%
Cancer-related molecular mechanisms research
0.94
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