Redox-dependent activation of protein kinase G1α contributes to transient receptor potential cation channel subfamily V member 1-mediated acute nociceptive pain behavior
Altogether, the findings of this study show the effects of redox-dependent PKG1α activation on capsaicin/TRPV1-mediated signaling in sensory neurons during acute nociceptive pain.
Authors
- Tobias Berg (ORCID: https://orcid.org/0000-0003-2668-0449)
- Achim Schmidtko (ORCID: https://orcid.org/0000-0001-7270-5075)
- Wiebke Kallenborn-Gerhardt
- Katharina Metzner
- Philip Eaton (ORCID: https://orcid.org/0000-0003-0748-2367)
- Elena Wang
- M. H. J. Koch (ORCID: https://orcid.org/0000-0003-0865-9590)
- Nabil Bahrami
Institutions
- Goethe University Frankfurt (DE)
- Queen Mary University of London (GB)
- William Harvey Research Institute (GB)
Publication Details
- Journal
- Redox Report
- Published
- 2025-08-25
- DOI
- https://doi.org/10.1080/13510002.2025.2549954
- Primary Topic
- Pain Mechanisms and Treatments
- Type
- article
- Field-Weighted Citation Impact
- 0.00
Funders
- Else Kröner-Fresenius-Stiftung