Poor Collateralization in T2DM: Role of SLC4A10+ MAIT Cells

These findings highlight the critical role of MAIT cells in regulating collateralization in type 2 diabetes chronic total occlusion patients and propose circulating MAIT cell levels as a potential biomarker for predicting and intervening in poor collateralization.

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Publication Details

Journal
Arteriosclerosis Thrombosis and Vascular Biology
Published
2025-08-14
DOI
https://doi.org/10.1161/atvbaha.125.322718
Primary Topic
Immune Cell Function and Interaction
Type
article
Field-Weighted Citation Impact
0.00
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article

Poor Collateralization in T2DM: Role of SLC4A10+ MAIT Cells

Shuai Chen, Qiujing Chen, Xue-Lian Wang, Fenghua Ding et al.
Arteriosclerosis Thrombosis and Vascular Biology
Immune Cell Function and Interaction
article

Poor Collateralization in T2DM: Role of SLC4A10+ MAIT Cells

Shuai Chen, Qiujing Chen, Xue-Lian Wang, Fenghua Ding, Lin Lu, Xiao Qun Wang, Xinrui Wu, Ting Zhang, Wenjia Li, Yang Dai, Zhiming Wu, Jingmeng Liu, Ruosen Yuan, Feifei Li, Ying Shen, Ke Huang, Yipaerguli Maimati
article en

Abstract

These findings highlight the critical role of MAIT cells in regulating collateralization in type 2 diabetes chronic total occlusion patients and propose circulating MAIT cell levels as a potential biomarker for predicting and intervening in poor collateralization.

Arteriosclerosis Thrombosis and Vascular BiologyVol. 45(10)
World Wide Web Consortium (US), First Affiliated Hospital Zhejiang University (CN)
Openalex Percentile: Top 78%
Immune Cell Function and Interaction
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Poor Collateralization in T2DM: Role of SLC4A10+ MAIT Cells — Shuai Chen, Qiujing Chen, et al. · Arteriosclerosis Thrombosis and Vascular Biology (2025) | TGRS Research Map | TGRS