1α, 25-dihydroxyvitamin D3 attenuates tumor necrosis factor-α-induced endothelial cell injury by modulating the tumor necrosis factor-α/nuclear factor kappa-B pathway
1α, 25(OH)2D3 regulates adhesion molecule expression in endothelial cells through the TNF-α/NF-κB pathway, laying the foundation for the clinical application of 1α, 25(OH)2D3 in the treatment of CV diseases.
Authors
- Yangyang Xia (ORCID: https://orcid.org/0000-0002-5754-0062)
- Qiuyuan Shao (ORCID: https://orcid.org/0000-0001-7314-1275)
- Qingyan Zhang (ORCID: https://orcid.org/0000-0003-0512-112X)
- Sixiu Liu (ORCID: https://orcid.org/0000-0001-7094-5892)
- Yuan Feng (ORCID: https://orcid.org/0009-0003-9317-1246)
- Bo Jin
Institutions
- Nanjing Drum Tower Hospital (CN)
Publication Details
- Journal
- CytoJournal
- Published
- 2025-08-01
- DOI
- https://doi.org/10.25259/cytojournal_220_2024
- Citations
- 1
- Primary Topic
- Vitamin D Research Studies
- Type
- article
- Field-Weighted Citation Impact
- 1.47