Study of NOX4/ROS/NF-κB signaling pathway in anti-ischemia-reperfusion injury effect and mechanism of STQJD

STQJD effectively ameliorates IS in MCAO/R rats by modulating the NOX4/TLR4/NF-κB/TNF-α signaling pathway, reducing inflammation and oxidative damage, and exerting neuroprotective effects.

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Publication Details

Journal
American Journal of Translational Research
Published
2025-01-01
DOI
https://doi.org/10.62347/mlzi7617
Primary Topic
Nitric Oxide and Endothelin Effects
Type
article
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article

Study of NOX4/ROS/NF-κB signaling pathway in anti-ischemia-reperfusion injury effect and mechanism of STQJD

Caiying Luo
American Journal of Translational Research
Nitric Oxide and Endothelin Effects
article

Study of NOX4/ROS/NF-κB signaling pathway in anti-ischemia-reperfusion injury effect and mechanism of STQJD

Caiying Luo
article en

Abstract

STQJD effectively ameliorates IS in MCAO/R rats by modulating the NOX4/TLR4/NF-κB/TNF-α signaling pathway, reducing inflammation and oxidative damage, and exerting neuroprotective effects.

American Journal of Translational ResearchVol. 17(7)
Yunnan University (CN), Kunming Medical University (CN)
Good health and well-being
Openalex Percentile: Top 76%
Nitric Oxide and Endothelin Effects
DOIOpen Access PDFOpenAlex
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Study of NOX4/ROS/NF-κB signaling pathway in anti-ischemia-reperfusion injury effect and mechanism of STQJD — Caiying Luo · American Journal of Translational Research (2025) | TGRS Research Map | TGRS