X inactive-specific transcript regulates mitochondrial function and neuronal differentiation of stem cells <i>via</i> IGF2BP2/CPT1A axis in models of spinal cord injury

These findings suggested that XIST modulated mitochondrial function and neural differentiation in NSCs through the IGF2BP2/CPT1A pathway. While preliminary <i>in vivo</i> results are encouraging, further studies are needed to determine the long-term therapeutic relevance and underlying mechanisms of XIST in SCI recovery.