COgnitive crisis: unveiling neurological consequences of carbon monoxide poisoning: a case report

Carbon Monoxide (CO) is commonly emitted by vehicles, industrial processes, and heating systems. It can lead to unintentional poisoning in enclosed spaces. Common findings include tachycardia, hypotension, hypoxemia, headache, dizziness, seizures, or coma. A thorough history is essential to suspect potential CO poisoning. Confirmatory tests include measuring carboxyhemoglobin levels in blood. The first step of management is to remove the patient from the source, followed by oxygen supplementation. In severe cases, hyperbaric oxygen therapy may be indicated. Prognosis depends on the level of exposure, as well as the promptness of treatment. Early intervention leads to better outcomes. Delayed neurological deficits are potential long-term outcomes. Prognosis for recovery improves significantly with the use of CO detectors in homes and education about the risks of CO exposure. We present a case of a 15-year-old male who presented with acute encephalopathy. Upon physical examination, laboratory study, and neuroimaging, a diagnosis of CO poisoning was made. The patient was intubated, placed on a ventilator, and managed conservatively with remarkable recovery.