CALR promotes corneal epithelial cell proliferation and migration through Wnt7a
CALR promotes proliferation and migration, inhibited senescence of HCE-2[50.B1] cells by Wnt7a, thus promoting corneal epithelial wound healing. This study will provide a theoretical basis for mechanism of CALR in corneal injury repair, and provide a new target for corneal injury clinical treatment.
Authors
- Lingling Wang (ORCID: https://orcid.org/0000-0003-0272-2974)
- Qian Li (ORCID: https://orcid.org/0000-0003-0920-4889)
- Ning Wei (ORCID: https://orcid.org/0000-0002-7790-7335)
Institutions
- Fourth People’s Hospital of Jinan (CN)
Publication Details
- Journal
- Molecular Biology Reports
- Published
- 2025-07-16
- DOI
- https://doi.org/10.1007/s11033-025-10810-x
- Citations
- 1
- Primary Topic
- Corneal Surgery and Treatments
- Type
- article
- Field-Weighted Citation Impact
- 1.36