Botulinum Toxin A Modulates Keratinocyte Proliferation and Inflammatory and Pruritic Mediators in Wound Healing
BTA promotes keratinocyte proliferation and reduces TGF-β-induced inflammatory and pruritus-associated mediators. These findings suggest that BTA may facilitate wound healing by promoting keratinocyte proliferation while simultaneously modulating inflammation and pruritic responses.
Authors
- Kwang Joong Kim (ORCID: https://orcid.org/0000-0003-4158-6100)
- SUNHAN SHIN (ORCID: https://orcid.org/0000-0002-7903-6879)
- Kwang Ho Kim (ORCID: https://orcid.org/0000-0001-5315-6031)
- Eun Joo Park (ORCID: https://orcid.org/0000-0002-9924-515X)
- Dayeon Jung (ORCID: https://orcid.org/0000-0003-3431-9397)
Institutions
- Hallym University (KR)
- Hallym University Sacred Heart Hospital (KR)
- Hallym University Medical Center (KR)
Publication Details
- Journal
- Annals of Dermatology
- Published
- 2025-01-01
- DOI
- https://doi.org/10.5021/ad.25.063
- Primary Topic
- Botulinum Toxin and Related Neurological Disorders
- Type
- article
- Field-Weighted Citation Impact
- 0.00
Funders
- National Research Foundation
- Hallym University
- National Research Foundation of Korea