The regulatory role of cell cycle exit and neuronal differentiation 1 in the pathogenesis of Parkinson’s disease by regulating the nuclear factor kappa B pathway

CEND1 plays a protective role in the Parkinson's disease cell model by suppressing NF-κB signaling pathway activation, offering a potential target and foundation for the diagnosis and therapy of Parkinson's disease.

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Publication Details

Journal
Neuroreport
Published
2025-07-01
DOI
https://doi.org/10.1097/wnr.0000000000002193
Primary Topic
Nuclear Receptors and Signaling
Type
article
Field-Weighted Citation Impact
0.00
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article

The regulatory role of cell cycle exit and neuronal differentiation 1 in the pathogenesis of Parkinson’s disease by regulating the nuclear factor kappa B pathway

Chan Li, Z Cong
Neuroreport
Nuclear Receptors and Signaling
article

The regulatory role of cell cycle exit and neuronal differentiation 1 in the pathogenesis of Parkinson’s disease by regulating the nuclear factor kappa B pathway

Chan Li, Z Cong
article en

Abstract

CEND1 plays a protective role in the Parkinson's disease cell model by suppressing NF-κB signaling pathway activation, offering a potential target and foundation for the diagnosis and therapy of Parkinson's disease.

NeuroreportVol. 36(13)
Tongren Hospital (CN), Wuhan Third Hospital (CN)
Good health and well-being
Openalex Percentile: Top 82%
Nuclear Receptors and Signaling
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The regulatory role of cell cycle exit and neuronal differentiation 1 in the pathogenesis of Parkinson’s disease by regulating the nuclear factor kappa B pathway — Chan Li, Z Cong · Neuroreport (2025) | TGRS Research Map | TGRS