Dcun1d3 is dispensable for spermatogenesis and male fertility in mice

These findings indicate that Dcun1d3 is not essential for spermatogenesis or male fertility in mice. This study provides evidence to streamline future investigations by excluding Dcun1d3 as a critical regulator of male germ cell development and offers useful insights for human fertility gene research.

Authors

Meng Liu (ORCID: https://orcid.org/0000-0002-6960-3163)

Institutions

Suzhou Municipal Hospital (CN)

Publication Details

Journal: American journal of clinical and experimental immunology.
Published: 2025-01-01
DOI: https://doi.org/10.62347/bzpe6333
Primary Topic: Nuclear Structure and Function
Type: article
Field-Weighted Citation Impact: 0.00

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article

Dcun1d3 is dispensable for spermatogenesis and male fertility in mice

Meng Liu

Jan 1, 2025

6 links

American journal of clinical and experimental immunology.

Nuclear Structure and Function

article

Dcun1d3 is dispensable for spermatogenesis and male fertility in mice

Meng Liu

January 1, 2025article en

Abstract

American journal of clinical and experimental immunology.Vol. 14(3)

Suzhou Municipal Hospital (CN)

Zero hunger

Openalex Percentile: Top 86%

Nuclear Structure and Function

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Dcun1d3 is dispensable for spermatogenesis and male fertility in mice — Meng Liu · American journal of clinical and experimental immunology. (2025) | TGRS Research Map | TGRS