S1PR4 promotes cell viability, invasion and glycolysis via the mTOR signaling pathway in endometriosis

In endometriosis, S1PR4 enhances cellular glycolysis by activating the mTOR signaling pathway, thereby promoting the viability and invasion of EESCs.

Authors

Qunxiu Lao
Zhiqin Xie (ORCID: https://orcid.org/0000-0002-3011-5031)
Cankun Zhou (ORCID: https://orcid.org/0000-0002-7253-6686)
Minqing Feng
Zheng Jianying
Xiaobin Huang (ORCID: https://orcid.org/0000-0002-6751-2495)
Rong Geng
H Yuan (ORCID: https://orcid.org/0000-0003-2137-8574)

Institutions

Foshan Maternity and Child Health Care Hospital (CN)

Publication Details

Journal: Gynecologic and Obstetric Investigation
Published: 2025-06-18
DOI: https://doi.org/10.1159/000546571
Primary Topic: Endometriosis Research and Treatment
Type: article
Field-Weighted Citation Impact: 0.00

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article

S1PR4 promotes cell viability, invasion and glycolysis via the mTOR signaling pathway in endometriosis

Qunxiu Lao, Zhiqin Xie, Cankun Zhou, Minqing Feng et al.

Jun 18, 2025

Gynecologic and Obstetric Investigation

Endometriosis Research and Treatment

article

S1PR4 promotes cell viability, invasion and glycolysis via the mTOR signaling pathway in endometriosis

Qunxiu Lao, Zhiqin Xie, Cankun Zhou, Minqing Feng, Zheng Jianying, Xiaobin Huang, Rong Geng, H Yuan

June 18, 2025article en

Abstract

In endometriosis, S1PR4 enhances cellular glycolysis by activating the mTOR signaling pathway, thereby promoting the viability and invasion of EESCs.

Gynecologic and Obstetric Investigation

Foshan Maternity and Child Health Care Hospital (CN)

Good health and well-being

Openalex Percentile: Top 80%

Endometriosis Research and Treatment

DOI Source OpenAlex

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S1PR4 promotes cell viability, invasion and glycolysis via the mTOR signaling pathway in endometriosis — Qunxiu Lao, Zhiqin Xie, et al. · Gynecologic and Obstetric Investigation (2025) | TGRS Research Map | TGRS