Does Endoplasmic Reticulum (ER) Stress Contribute to T-cell Exhaustion in B-ALL?
Collectively, our gene expression data suggest ER stress activation in CD8+ T lymphocytes from B-ALL patients. These findings warrant further investigation into ER stress-related signaling pathways and their potential role in promoting T-cell exhaustion in B-ALL.
Authors
- Hossein Karami (ORCID: https://orcid.org/0000-0001-9765-776X)
- Ahmad Najafi (ORCID: https://orcid.org/0000-0001-9821-336X)
- Armin Akbar (ORCID: https://orcid.org/0000-0002-8822-218X)
- Hossein Asgarian‐Omran (ORCID: https://orcid.org/0000-0003-3816-8792)
- Alireza Karimi (ORCID: https://orcid.org/0000-0002-3535-3952)
- Mohammad Naderisorki (ORCID: https://orcid.org/0000-0001-8638-4057)
- Amir Kahrizi (ORCID: https://orcid.org/0009-0001-5740-3543)
- Mohsen Tehrani (ORCID: https://orcid.org/0000-0002-9292-4520)
Institutions
- Imam Khomeini Hospital (IR)
- Mazandaran University of Medical Sciences (IR)
Publication Details
- Journal
- PubMed
- Published
- 2025-05-26
- DOI
- https://doi.org/10.22034/iji.2025.105453.2949
- Primary Topic
- Acute Lymphoblastic Leukemia research
- Type
- article
- Field-Weighted Citation Impact
- 0.00