Abstract ND04: ABP-102/CT-P72: a novel HER2 x CD3 T cell engager with selective activity for HER2-overexpressing tumors and reduced activity on cells with normal HER2 expression levels

Abstract HER2 is overexpressed in breast, gastric, and other cancers, with limited expression in normal tissues. Impactful HER2-targeted therapeutics include monoclonal antibodies, tyrosine kinase inhibitors, and antibody-drug conjugates. However, resistance remains an issue. Bispecific antibody T cell engagers (TCEs) have unrealized potential as novel treatments for solid tumors, with notable past successes in hematological malignancies and the encouraging recent FDA approval of tarlatamab for a solid tumor indication. Previous attempts to develop HER2 TCEs have met with toxicity issues. To develop a safer agent, we designed ABP-102/CT-P72, a next-generation HER2 x CD3 tetravalent bispecific (TetraBi) IgG1-[L]-scFv format antibody with selective activity against HER2-overexpressing tumor cells through the reduction of the affinity of each of the bivalent HER2 binding arms to reduce activity against HER2-low cells. ABP-102/CT-P72 is engineered to reduce the potential for on-target, off-tumor toxicity in normal tissues with low HER2 expression, and its IgG-[L]-scFv format features functionally monovalent CD3 binding for T cell engagement. Pre-clinical studies of in vitro activity as well as in vivo efficacy and safety of ABP-102 were performed. In vitro, ABP-102/CT-P72-mediated T cell activation, cytotoxicity, and cytokine release occurred in a HER2-expression level dependent manner. In vivo studies were done using xenograft tumor models implanted with human HER2-expressing cells, followed by application of human PBMCs. ABP-102/CT-P72 demonstrated potent tumor growth inhibition in HER2-overexpressing models (NCI-N87 and BT-474), with reduced tumor growth inhibition in a HER2-low model (HT55), as expected. In the HER2-overexpressing models, ABP-102/CT-P72 displayed up to a two-fold increased tumor growth inhibition compared to a biosimilar of runimotamab, a HER2 x CD3 TCE. ABP-102/CT-P72 was also well tolerated in cynomolgus macaques. Given its selectivity for HER2-overexpressing tumor cells, ABP-102/CT-P72 represents a novel therapeutic approach with the potential to overcome the limitations of current agents by reducing toxicity in HER2-expressing normal tissues and sparing cells with endogenous levels of HER2. With preclinical studies of ABP-102/CT-P72 demonstrating potent efficacy and safety, we expect that this will allow for an expanded therapeutic window in upcoming clinical trials. Abpro and CELLTRION, INC. are co-developing ABP-102, which CELLTRION, INC. has designated under the project code name CT-P72. Citation Format: Adam Pelzek, Shaun Murphy, Sanam Ebtehaj, Marco Muda, James Lulo, Benjamin Barros, Lucy Zhang, Chaohua Zhang, Robert Markelewicz, Bobin Kang, Juhyung Seok, Bokhyeon Im, Riri Kwon, Hyunkyu Choi, Ji-Seon Kang, Jungsun Ahn, Eun Yeong Shim, YeonJung Kim, Jiyoung Shin. ABP-102/CT-P72: a novel HER2 x CD3 T cell engager with selective activity for HER2-overexpressing tumors and reduced activity on cells with normal HER2 expression levels [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2025; Part 2 (Late-Breaking, Clinical Trial, and Invited Abstracts); 2025 Apr 25-30; Chicago, IL. Philadelphia (PA): AACR; Cancer Res 2025;85(8_Suppl_2):Abstract nr ND04.

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Journal
Cancer Research
Published
2025-05-22
DOI
https://doi.org/10.1158/1538-7445.am2025-nd04
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1
Primary Topic
CAR-T cell therapy research
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article
Field-Weighted Citation Impact
1.05
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article

Abstract ND04: ABP-102/CT-P72: a novel HER2 x CD3 T cell engager with selective activity for HER2-overexpressing tumors and reduced activity on cells with normal HER2 expression levels

Marco Muda, YeonJung Kim, Shaun Murphy, Jeong Ho Seok et al.
1 citations
Cancer Research
CAR-T cell therapy research
1.05
article

Abstract ND04: ABP-102/CT-P72: a novel HER2 x CD3 T cell engager with selective activity for HER2-overexpressing tumors and reduced activity on cells with normal HER2 expression levels

Marco Muda, YeonJung Kim, Shaun Murphy, Jeong Ho Seok, Hyeji Choi, Lucy Zhang, Jungmin Ahn, James Lulo, Eun Yeong Shim, Chaohua Zhang, Bobin Kang, Sanam Ebtehaj, Riri Kwon, Robert J Markelewicz, Adam J. Pelzek, Ji-Young Shin, Ji-Seon Kang, Burr Im, B. de A. Barros
article en
1 citations

Abstract

Abstract HER2 is overexpressed in breast, gastric, and other cancers, with limited expression in normal tissues. Impactful HER2-targeted therapeutics include monoclonal antibodies, tyrosine kinase inhibitors, and antibody-drug conjugates. However, resistance remains an issue. Bispecific antibody T cell engagers (TCEs) have unrealized potential as novel treatments for solid tumors, with notable past successes in hematological malignancies and the encouraging recent FDA approval of tarlatamab for a solid tumor indication. Previous attempts to develop HER2 TCEs have met with toxicity issues. To develop a safer agent, we designed ABP-102/CT-P72, a next-generation HER2 x CD3 tetravalent bispecific (TetraBi) IgG1-[L]-scFv format antibody with selective activity against HER2-overexpressing tumor cells through the reduction of the affinity of each of the bivalent HER2 binding arms to reduce activity against HER2-low cells. ABP-102/CT-P72 is engineered to reduce the potential for on-target, off-tumor toxicity in normal tissues with low HER2 expression, and its IgG-[L]-scFv format features functionally monovalent CD3 binding for T cell engagement. Pre-clinical studies of in vitro activity as well as in vivo efficacy and safety of ABP-102 were performed. In vitro, ABP-102/CT-P72-mediated T cell activation, cytotoxicity, and cytokine release occurred in a HER2-expression level dependent manner. In vivo studies were done using xenograft tumor models implanted with human HER2-expressing cells, followed by application of human PBMCs. ABP-102/CT-P72 demonstrated potent tumor growth inhibition in HER2-overexpressing models (NCI-N87 and BT-474), with reduced tumor growth inhibition in a HER2-low model (HT55), as expected. In the HER2-overexpressing models, ABP-102/CT-P72 displayed up to a two-fold increased tumor growth inhibition compared to a biosimilar of runimotamab, a HER2 x CD3 TCE. ABP-102/CT-P72 was also well tolerated in cynomolgus macaques. Given its selectivity for HER2-overexpressing tumor cells, ABP-102/CT-P72 represents a novel therapeutic approach with the potential to overcome the limitations of current agents by reducing toxicity in HER2-expressing normal tissues and sparing cells with endogenous levels of HER2. With preclinical studies of ABP-102/CT-P72 demonstrating potent efficacy and safety, we expect that this will allow for an expanded therapeutic window in upcoming clinical trials. Abpro and CELLTRION, INC. are co-developing ABP-102, which CELLTRION, INC. has designated under the project code name CT-P72. Citation Format: Adam Pelzek, Shaun Murphy, Sanam Ebtehaj, Marco Muda, James Lulo, Benjamin Barros, Lucy Zhang, Chaohua Zhang, Robert Markelewicz, Bobin Kang, Juhyung Seok, Bokhyeon Im, Riri Kwon, Hyunkyu Choi, Ji-Seon Kang, Jungsun Ahn, Eun Yeong Shim, YeonJung Kim, Jiyoung Shin. ABP-102/CT-P72: a novel HER2 x CD3 T cell engager with selective activity for HER2-overexpressing tumors and reduced activity on cells with normal HER2 expression levels [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2025; Part 2 (Late-Breaking, Clinical Trial, and Invited Abstracts); 2025 Apr 25-30; Chicago, IL. Philadelphia (PA): AACR; Cancer Res 2025;85(8_Suppl_2):Abstract nr ND04.

Cancer ResearchVol. 85(8_Supplement_2)
Celltrion (South Korea) (KR)
Openalex Percentile: Top 22%
CAR-T cell therapy research
1.05
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