Self-propagating wave drives morphogenesis of skull bones in vivo

Cellular motion is a key feature of tissue morphogenesis and is often driven by migration. However, migration need not explain cell motion in contexts where there is little free space or no obvious substrate, such as those found during organogenesis of mesenchymal organs including the embryonic skull. Through ex vivo imaging, biophysical modeling, and perturbation experiments, we find that mechanical feedback between cell fate and stiffness drives bone expansion and controls bone size in vivo. This mechanical feedback system is sufficient to propagate a wave of differentiation that establishes a collagen gradient which we find sufficient to describe patterns of osteoblast motion. Our work provides a mechanism for coordinated motion that may not rely upon cell migration but on emergent properties of the mesenchymal collective. Identification of such alternative mechanisms of mechanochemical coupling between differentiation and morphogenesis will help in understanding how directed cellular motility arises in complex environments with inhomogeneous material properties.

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Publication Details

Journal
Nature Communications
Published
2025-05-09
DOI
https://doi.org/10.1038/s41467-025-59164-9
Citations
4
Primary Topic
Cellular Mechanics and Interactions
Type
article
Field-Weighted Citation Impact
4.61

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article

Self-propagating wave drives morphogenesis of skull bones in vivo

Adrian Lahola-Chomiak, Jacqueline M. Tabler, Johanna Lattner, Elke Ulbricht et al.
4 citations
Nature Communications
Cellular Mechanics and Interactions
4.61
article

Self-propagating wave drives morphogenesis of skull bones in vivo

Adrian Lahola-Chomiak, Jacqueline M. Tabler, Johanna Lattner, Elke Ulbricht, Anna Taubenberger, Yiteng Dang, Steffen Rulands, Diana Alves Afonso
article en
4 citations

Abstract

Cellular motion is a key feature of tissue morphogenesis and is often driven by migration. However, migration need not explain cell motion in contexts where there is little free space or no obvious substrate, such as those found during organogenesis of mesenchymal organs including the embryonic skull. Through ex vivo imaging, biophysical modeling, and perturbation experiments, we find that mechanical feedback between cell fate and stiffness drives bone expansion and controls bone size in vivo. This mechanical feedback system is sufficient to propagate a wave of differentiation that establishes a collagen gradient which we find sufficient to describe patterns of osteoblast motion. Our work provides a mechanism for coordinated motion that may not rely upon cell migration but on emergent properties of the mesenchymal collective. Identification of such alternative mechanisms of mechanochemical coupling between differentiation and morphogenesis will help in understanding how directed cellular motility arises in complex environments with inhomogeneous material properties.

Nature CommunicationsVol. 16(1)
Max Planck Institute for the Physics of Complex Systems (DE), Center for Systems Biology Dresden (DE), Max Planck Institute of Molecular Cell Biology and Genetics (DE), Ludwig-Maximilians-Universität München (DE)
Deutsche Forschungsgemeinschaft, National Institutes of Health, National Institute of Dental and Craniofacial Research
Openalex Percentile: Top 6%
Cellular Mechanics and Interactions
4.61
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