m6A hypermethylation of TCF-1 regulated by METTL16 promotes acute myeloid leukemia

Collectively, METTL16 deficiency promoted the persistence of CAR-T cells and memory formation in AML. Therefore, targeting METTL16 may stimulate the anti-tumor immunity in AML.

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Publication Details

Journal
Clinical and Experimental Medicine
Published
2025-04-29
DOI
https://doi.org/10.1007/s10238-025-01669-0
Citations
3
Primary Topic
RNA modifications and cancer
Type
article
Field-Weighted Citation Impact
2.42
Controls
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article

m6A hypermethylation of TCF-1 regulated by METTL16 promotes acute myeloid leukemia

Jingyi Li, Hui Kang
3 citations
Clinical and Experimental Medicine
RNA modifications and cancer
2.42
article

m6A hypermethylation of TCF-1 regulated by METTL16 promotes acute myeloid leukemia

Jingyi Li, Hui Kang
article en
3 citations

Abstract

Collectively, METTL16 deficiency promoted the persistence of CAR-T cells and memory formation in AML. Therefore, targeting METTL16 may stimulate the anti-tumor immunity in AML.

Clinical and Experimental MedicineVol. 25(1)
Taiyuan Maternity and Child Care Hospital (CN)
Good health and well-being
Openalex Percentile: Top 12%
RNA modifications and cancer
2.42
DOIOpen Access PDFOpenAlex
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m6A hypermethylation of TCF-1 regulated by METTL16 promotes acute myeloid leukemia — Jingyi Li, Hui Kang · Clinical and Experimental Medicine (2025) | TGRS Research Map | TGRS