Abstract 901: KLF4-induced melanocyte-schwann plasticity promotes immune escape in melanoma via LAG3 checkpoint

Abstract Precision therapy is crucial to the successful treatment of rare tumors with limited treatment options. Here, we report a rare case of primary anterior mediastinal melanoma with liver metastasis. The patient had previously failed anti-PD1 treatment, leading us to profile the molecular features of the tumor to identify alternative therapeutic targets. Through comprehensive multi-omics analyses, we discovered a high degree of tumor cell plasticity in this rare case, with a subset of tumor cells displaying a Schwann cell phenotype evolved from melanocyte tumor cells. Cell-cell communication analysis revealed that the Schwann-like tumor cells may evade immune surveillance through the LAG3-LGALS3 immune checkpoint. The patient was then recommended for enrollment in a trial using combination therapy with anti-PD1 and anti-LAG3, and the disease remained stable for up to 24 months. Further investigation into the underlying mechanism showed that transcription factor KLF4-mediated transcription reprogramming drives B16 melanoma cell trans-differentiation into a Schwann cell phenotype. This trans-differentiation sensitizes the tumor cells to anti-LAG3 therapy. By analyzing melanoma tissue microarrays and external single-cell sequencing data, we found that this melanocyte-Schwann plasticity (MSP) existed in around 20% of melanoma cases. Moreover, a public immunotherapy cohort and our data indicate that MSP is associated with poor response to anti-PD1 treatment, suggesting that MSP may serve as a predictive biomarker for immunotherapy efficacy. Overall, our study not only provides a paradigm for precision immunotherapy in the context of rare tumors, but also reveals a potentially effective immunotherapeutic strategy for advanced melanoma with melanocyte-Schwann plasticity. Citation Format: An Zhao, Xuefei Liu, Zhixiang Zuo. KLF4-induced melanocyte-schwann plasticity promotes immune escape in melanoma via LAG3 checkpoint [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2025; Part 1 (Regular Abstracts); 2025 Apr 25-30; Chicago, IL. Philadelphia (PA): AACR; Cancer Res 2025;85(8_Suppl_1):Abstract nr 901.

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Journal
Cancer Research
Published
2025-04-21
DOI
https://doi.org/10.1158/1538-7445.am2025-901
Primary Topic
Kruppel-like factors research
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article
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article

Abstract 901: KLF4-induced melanocyte-schwann plasticity promotes immune escape in melanoma via LAG3 checkpoint

An Zhao, Zhixiang Zuo, Xuefei Liu
Cancer Research
Kruppel-like factors research
article

Abstract 901: KLF4-induced melanocyte-schwann plasticity promotes immune escape in melanoma via LAG3 checkpoint

An Zhao, Zhixiang Zuo, Xuefei Liu
article en

Abstract

Abstract Precision therapy is crucial to the successful treatment of rare tumors with limited treatment options. Here, we report a rare case of primary anterior mediastinal melanoma with liver metastasis. The patient had previously failed anti-PD1 treatment, leading us to profile the molecular features of the tumor to identify alternative therapeutic targets. Through comprehensive multi-omics analyses, we discovered a high degree of tumor cell plasticity in this rare case, with a subset of tumor cells displaying a Schwann cell phenotype evolved from melanocyte tumor cells. Cell-cell communication analysis revealed that the Schwann-like tumor cells may evade immune surveillance through the LAG3-LGALS3 immune checkpoint. The patient was then recommended for enrollment in a trial using combination therapy with anti-PD1 and anti-LAG3, and the disease remained stable for up to 24 months. Further investigation into the underlying mechanism showed that transcription factor KLF4-mediated transcription reprogramming drives B16 melanoma cell trans-differentiation into a Schwann cell phenotype. This trans-differentiation sensitizes the tumor cells to anti-LAG3 therapy. By analyzing melanoma tissue microarrays and external single-cell sequencing data, we found that this melanocyte-Schwann plasticity (MSP) existed in around 20% of melanoma cases. Moreover, a public immunotherapy cohort and our data indicate that MSP is associated with poor response to anti-PD1 treatment, suggesting that MSP may serve as a predictive biomarker for immunotherapy efficacy. Overall, our study not only provides a paradigm for precision immunotherapy in the context of rare tumors, but also reveals a potentially effective immunotherapeutic strategy for advanced melanoma with melanocyte-Schwann plasticity. Citation Format: An Zhao, Xuefei Liu, Zhixiang Zuo. KLF4-induced melanocyte-schwann plasticity promotes immune escape in melanoma via LAG3 checkpoint [abstract]. In: Proceedings of the American Association for Cancer Research Annual Meeting 2025; Part 1 (Regular Abstracts); 2025 Apr 25-30; Chicago, IL. Philadelphia (PA): AACR; Cancer Res 2025;85(8_Suppl_1):Abstract nr 901.

Cancer ResearchVol. 85(8_Supplement_1)
Sun Yat-sen University (CN), Chinese Academy of Sciences (CN), Zhejiang Cancer Hospital (CN), Sun Yat-sen University Cancer Center (CN)
Good health and well-being
Openalex Percentile: Top 93%
Kruppel-like factors research
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