OGA promotes human dental pulp stem cell senescence and inhibits mitophagy by inhibition of O-GlcNAcylation of KLF2

Silencing of OGA promotes mitophagy and inhibits DPSC senescence by promoting the O-GlcNAcylation of KLF2, suggesting a novel mechanism underlying DPSC senescence.

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Publication Details

Journal
BMC Oral Health
Published
2025-04-18
DOI
https://doi.org/10.1186/s12903-025-05927-1
Citations
1
Primary Topic
Kruppel-like factors research
Type
article
Field-Weighted Citation Impact
0.81
Controls
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article

OGA promotes human dental pulp stem cell senescence and inhibits mitophagy by inhibition of O-GlcNAcylation of KLF2

Yanyan Ding, Yan Ran
1 citations
BMC Oral Health
Kruppel-like factors research
0.81
article

OGA promotes human dental pulp stem cell senescence and inhibits mitophagy by inhibition of O-GlcNAcylation of KLF2

Yanyan Ding, Yan Ran
article en
1 citations

Abstract

Silencing of OGA promotes mitophagy and inhibits DPSC senescence by promoting the O-GlcNAcylation of KLF2, suggesting a novel mechanism underlying DPSC senescence.

BMC Oral HealthVol. 25(1)
Ningbo No.6 Hospital (CN)
Zero hunger
Openalex Percentile: Top 29%
Kruppel-like factors research
0.81
DOIOpen Access PDFOpenAlex
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OGA promotes human dental pulp stem cell senescence and inhibits mitophagy by inhibition of O-GlcNAcylation of KLF2 — Yanyan Ding, Yan Ran · BMC Oral Health (2025) | TGRS Research Map | TGRS