Nitrosation of CD36 Regulates Endothelial Function and Serum Lipids

This work suggests that CD36 nitrosation occurs as a protective mechanism to prevent EC lipotoxicity.

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Publication Details

Journal
Arteriosclerosis Thrombosis and Vascular Biology
Published
2025-04-17
DOI
https://doi.org/10.1161/atvbaha.124.321964
Citations
5
Primary Topic
Caveolin-1 and cellular processes
Type
article
Field-Weighted Citation Impact
5.76
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article

Nitrosation of CD36 Regulates Endothelial Function and Serum Lipids

Ilya Levental, Xiaohong Shu, Linda Columbus, Skylar A. Loeb et al.
5 citations
Arteriosclerosis Thrombosis and Vascular Biology
Caveolin-1 and cellular processes
5.76
article

Nitrosation of CD36 Regulates Endothelial Function and Serum Lipids

Ilya Levental, Xiaohong Shu, Linda Columbus, Skylar A. Loeb, Ray Mitchell, Melissa A. Luse, T.C. Stevenson Keller, Timothy M. Sveeggen, Richard D. Minshall, Wyatt J. Schug, Brant E. Isakson, Claire A. Ruddiman, Pooneh Bagher, Kandice R. Levental, Norbert Leitinger, Miriam M. Cortese‐Krott, Anna Kosmach, Luke S. Dunaway, Shruthi Nyshadham, Caitlin M. Pavelec, Rachel Tessema, Alicia Carvalho
article en
5 citations

Abstract

This work suggests that CD36 nitrosation occurs as a protective mechanism to prevent EC lipotoxicity.

Arteriosclerosis Thrombosis and Vascular BiologyVol. 45(7)
Dalian Medical University (CN), University of Illinois Chicago (US), University of Chicago (US), Anna Needs Neuroblastoma Answers (US), Cardiovascular Research Center (BR), Norwegian Womens Public Health Association (NO), Heinrich Heine University Düsseldorf (DE), University of Virginia (US), University of Nebraska Medical Center (US)
Good health and well-being
Openalex Percentile: Top 4%
Caveolin-1 and cellular processes
5.76
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