Omics Analyses Uncover Host Networks Defining Virus-Permissive and -Hostile Cellular States

The capacity of host cells to sustain or restrict virus infection is influenced by their proteome. Understanding the compendium of proteins defining cellular permissiveness is key to many questions in fundamental virology. Here, we apply a multi-omic approach to determine the proteins that are associated with highly permissive, intermediate, and hostile cellular states. We observed two groups of differentially regulated genes: (i) with robust changes in mRNA and protein levels and (ii) with protein/RNA discordances. While many of the latter are classified as interferon-stimulated genes (ISGs), most exhibit no antiviral effects in overexpression screens. This suggests that IFN-dependent protein changes can be better indicators of antiviral function than mRNA levels. Phosphoproteomics revealed an additional regulatory layer involving non-signaling proteins with altered phosphorylation. Indeed, we confirmed that several permissiveness-associated proteins with changes in abundance or phosphorylation regulate infection fitness. Altogether, our study provides a comprehensive and systematic map of the cellular alterations driving virus susceptibility.

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Publication Details

Journal
Molecular & Cellular Proteomics
Published
2025-04-08
DOI
https://doi.org/10.1016/j.mcpro.2025.100966
Citations
7
Primary Topic
Viral Infections and Immunology Research
Type
article
Field-Weighted Citation Impact
9.72

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article

Omics Analyses Uncover Host Networks Defining Virus-Permissive and -Hostile Cellular States

Sam J. Wilson, Quan Gu, Massimo Palmarini, Alfredo Castelló et al.
7 citations
Molecular & Cellular Proteomics
Viral Infections and Immunology Research
9.72
article

Omics Analyses Uncover Host Networks Defining Virus-Permissive and -Hostile Cellular States

Sam J. Wilson, Quan Gu, Massimo Palmarini, Alfredo Castelló, Sabrina Liberatori, Shabaz Mohammed, David L. Robertson, Honglin Chen, Philip D. Charles
article en
7 citations

Abstract

The capacity of host cells to sustain or restrict virus infection is influenced by their proteome. Understanding the compendium of proteins defining cellular permissiveness is key to many questions in fundamental virology. Here, we apply a multi-omic approach to determine the proteins that are associated with highly permissive, intermediate, and hostile cellular states. We observed two groups of differentially regulated genes: (i) with robust changes in mRNA and protein levels and (ii) with protein/RNA discordances. While many of the latter are classified as interferon-stimulated genes (ISGs), most exhibit no antiviral effects in overexpression screens. This suggests that IFN-dependent protein changes can be better indicators of antiviral function than mRNA levels. Phosphoproteomics revealed an additional regulatory layer involving non-signaling proteins with altered phosphorylation. Indeed, we confirmed that several permissiveness-associated proteins with changes in abundance or phosphorylation regulate infection fitness. Altogether, our study provides a comprehensive and systematic map of the cellular alterations driving virus susceptibility.

Molecular & Cellular ProteomicsVol. 24(5)
Open Data Institute (GB), University of Oxford (GB), Science Oxford (GB), MRC University of Glasgow Centre for Virus Research (GB), MRC Epidemiology Unit (GB)
University of Oxford, Medical Research Council, Engineering and Physical Sciences Research Council, FP7 Research infrastructures
Openalex Percentile: Top 2%
Viral Infections and Immunology Research
9.72
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