The Mechanism of Xuanyu Tongjing Decoction Regulating NOD/NFκB Pathway to Inhibit Ectopic Tissue Inflammation to Reduce Ovarian Damage in Rats with Ovarian Endometriosis

In rats with proliferative OEM, XYTJD may down-regulate the NOD/NFκB pathway in ectopic tissues, consequently alleviating ovarian tissue damage by reducing inflammation brought on by ectopic tissues.

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Publication Details

Journal
Drug Design Development and Therapy
Published
2025-04-01
DOI
https://doi.org/10.2147/dddt.s500129
Citations
1
Primary Topic
Endometriosis Research and Treatment
Type
article
Field-Weighted Citation Impact
1.70
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article

The Mechanism of Xuanyu Tongjing Decoction Regulating NOD/NFκB Pathway to Inhibit Ectopic Tissue Inflammation to Reduce Ovarian Damage in Rats with Ovarian Endometriosis

Fengting Zhai, Guiping Hu, Weisen Fan, Li Wang et al.
1 citations
Drug Design Development and Therapy
Endometriosis Research and Treatment
1.70
article

The Mechanism of Xuanyu Tongjing Decoction Regulating NOD/NFκB Pathway to Inhibit Ectopic Tissue Inflammation to Reduce Ovarian Damage in Rats with Ovarian Endometriosis

Fengting Zhai, Guiping Hu, Weisen Fan, Li Wang, Zheng Yuan
article en
1 citations

Abstract

In rats with proliferative OEM, XYTJD may down-regulate the NOD/NFκB pathway in ectopic tissues, consequently alleviating ovarian tissue damage by reducing inflammation brought on by ectopic tissues.

Drug Design Development and TherapyVol. Volume 19
Shandong University of Traditional Chinese Medicine (CN), Chinese Academy of Medical Sciences & Peking Union Medical College (CN), Guang’anmen Hospital (CN), Affiliated Hospital of Shandong University of Traditional Chinese Medicine (CN)
Zero hunger
Openalex Percentile: Top 18%
Endometriosis Research and Treatment
1.70
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The Mechanism of Xuanyu Tongjing Decoction Regulating NOD/NFκB Pathway to Inhibit Ectopic Tissue Inflammation to Reduce Ovarian Damage in Rats with Ovarian Endometriosis — Fengting Zhai, Guiping Hu, et al. · Drug Design Development and Therapy (2025) | TGRS Research Map | TGRS