On the question of cerebral venous dyscirculation syndrome

The problem of cerebral venous dyscirculation, the development of cerebral venous stagnation is relevant. This can lead to the development of cephalalgic syndrome at night and in the morning, the development of intracranial hypertension due to difficulty with the resorption of cerebrospinal fluid in the cerebral sinuses, acute disruption of venous cerebral circulation, specifically acute venous hemorrhage and venous cerebral infarction. Objective — to clarify the features of dysfunction of venous cerebral blood flow in patients who have suffered a closed head injury. Materials and methods. 30 patients with signs of cerebral venous congestion who had undergone a closed head injury were examined using encephaloangioscintigraphy in the vascular pools of the hemispheres (cerebral veins) and sinuses (transverse sinuses) Results and discussion. Based on encephaloangioscintigraphy studies, with computer processing of data and determination of the cerebral venous outflow criterion T1/2, the following results were obtained: left hemisphere — T1/2 = (7.80 ± 0.44) s, right hemisphere T1/2 = (7.60 ± 0.42) s, left transverse sinus T1/2 = (8.10 ± 0.46) s, right transverse sinus T1/2 = (7.80 ± 0.43) s. This indicates a significant simultaneous increase in the T1/2 factor in the vascular pools of the hemispheres and transverse sinuses in patients with cerebral venous congestion, compared with data from the control group, approximately twofold increase. Conclusions. In patients with cerebral venous congestion who have experienced a closed head injury, encephaloangioscintigraphy reveals an approximately twofold inhibition of venous cerebral blood flow in the vascular basins of the hemispheres (cerebral veins) and transverse sinuses. The latter indicates dysfunction of the central neurogenic circuit of regulation. Therefore, it is advisable to identify cerebral venous dyscirculation syndrome.