Germline copy number variants in RUNX1: An updated case report and a decade-old red herring

Pathogenic/likely pathogenic (P/LP) germline variants in RUNX1 cause familial platelet disorder with associated myeloid malignancies (FPDMM), also known as RUNX1-Familial Platelet Disorder (RUNX1-FPD, or FPD), a condition characterized by qualitative and quantitative platelet defects and predisposition to hematopoietic malignancies. Here, we present follow up to a case of a woman with acute myeloid leukemia and lifelong thrombocytopenia which had previously been attributed to presumptive pathogenic (P) GATA2 missense variants. However, re-evaluation with updated molecular technology sensitive for detection of copy number variants (CNVs) led to the identification of a P deletion of exons 5-6 in RUNX1, which had been undetected when examined at first presentation. This case highlights the importance of comprehensive molecular evaluation and careful variant interpretation, especially regarding CNVs.

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Journal
BJC Reports
Published
2025-03-27
DOI
https://doi.org/10.1038/s44276-024-00117-y
Primary Topic
Acute Myeloid Leukemia Research
Type
article
Field-Weighted Citation Impact
0.00

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article

Germline copy number variants in RUNX1: An updated case report and a decade-old red herring

Shawn Chong, Marshall S. Horwitz, David J. Young, Hamish S. Scott et al.
BJC Reports
Acute Myeloid Leukemia Research
article

Germline copy number variants in RUNX1: An updated case report and a decade-old red herring

Shawn Chong, Marshall S. Horwitz, David J. Young, Hamish S. Scott, Natalie Deuitch, K. L. Craft, Lucy A. Godley, Erica Bresciani, Paul Liu, Amra Kajdic
article en

Abstract

Pathogenic/likely pathogenic (P/LP) germline variants in RUNX1 cause familial platelet disorder with associated myeloid malignancies (FPDMM), also known as RUNX1-Familial Platelet Disorder (RUNX1-FPD, or FPD), a condition characterized by qualitative and quantitative platelet defects and predisposition to hematopoietic malignancies. Here, we present follow up to a case of a woman with acute myeloid leukemia and lifelong thrombocytopenia which had previously been attributed to presumptive pathogenic (P) GATA2 missense variants. However, re-evaluation with updated molecular technology sensitive for detection of copy number variants (CNVs) led to the identification of a P deletion of exons 5-6 in RUNX1, which had been undetected when examined at first presentation. This case highlights the importance of comprehensive molecular evaluation and careful variant interpretation, especially regarding CNVs.

BJC ReportsVol. 3(1)
South Australia Pathology (AU), Northwestern University (US), University of South Australia (AU), University of Washington (US), National Human Genome Research Institute (US), National Heart, Lung, and Blood Institute (US), Centre for Cancer Biology (AU)
Cancer Council South Australia, National Heart, Lung, and Blood Institute, National Human Genome Research Institute
Good health and well-being
Openalex Percentile: Top 92%
Acute Myeloid Leukemia Research
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Germline copy number variants in RUNX1: An updated case report and a decade-old red herring — Shawn Chong, Marshall S. Horwitz, et al. · BJC Reports (2025) | TGRS Research Map | TGRS