CMTM3 regulates vascular endothelial cell dysfunction by influencing pulmonary vascular endothelial permeability and inflammation in ARDS
These findings demonstrated that CMTM3 played a critical role in the development of ARDS by influencing permeability of the pulmonary vascular endothelial cells and lung inflammation. Therefore, CMTM3 is a potential therapeutic target in ARDS.
Authors
- Lihe Chen (ORCID: https://orcid.org/0000-0002-6207-6364)
- Haiyan Xue (ORCID: https://orcid.org/0000-0002-8600-9270)
- Gang Zhou
- Z D Wang
- Chun Fu (ORCID: https://orcid.org/0000-0001-6947-1159)
- Fengxue Zhu (ORCID: https://orcid.org/0009-0008-8715-4059)
- Xiujuan Zhao
- Ziyan Xiao (ORCID: https://orcid.org/0009-0000-8364-8037)
- Shu Li (ORCID: https://orcid.org/0000-0001-8409-5390)
Institutions
- Peking University (CN)
- Peking University People's Hospital (CN)
Publication Details
- Journal
- Frontiers in Immunology
- Published
- 2025-03-24
- DOI
- https://doi.org/10.3389/fimmu.2025.1544610
- Citations
- 3
- Primary Topic
- Respiratory Support and Mechanisms
- Type
- article
- Field-Weighted Citation Impact
- 4.56