Selective Autophagy Mediated by Protein Ubiquitination in Major Prevalent Zoonoses

Zoonotic diseases not only cause great harm to animal health but also involve the development of animal husbandry, which in turn endangers human life and health and public health safety. Protein ubiquitination and autophagy are important ways for the body to degrade invading pathogens, which correspond to the ubiquitin (Ub)-proteasome system (UPS) and autophagic lysosomal pathway (ALP), respectively, and play an important role in the occurrence and development of diseases. For UPS, the substrate is delivered to the 26S proteasome system via a ubiquitination cascade and subsequently degraded and removed. For ALP, the substrate is encapsulated to form autophagosomes, which subsequently fuse with lysosomes to form autophagolysosomes, which are eventually degraded and cleared. However, a variety of zoonotic pathogens can interfere with the protein ubiquitination pathway and autophagy process to promote self-replication and survival, and resist host immune defense. This article reviews the mechanisms by which multiple pathogens interfere with protein degradation pathways, providing a new perspective for the treatment and prevention of zoonotic diseases.

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Publication Details

Journal
Transboundary and Emerging Diseases
Published
2025-01-01
DOI
https://doi.org/10.1155/tbed/6238787
Primary Topic
Toxoplasma gondii Research Studies
Type
article
Field-Weighted Citation Impact
0.00

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article

Selective Autophagy Mediated by Protein Ubiquitination in Major Prevalent Zoonoses

Yuefeng Chu, Lingjie Wang, Hanwei Jiao, Jixiang Li et al.
Transboundary and Emerging Diseases
Toxoplasma gondii Research Studies
article

Selective Autophagy Mediated by Protein Ubiquitination in Major Prevalent Zoonoses

Yuefeng Chu, Lingjie Wang, Hanwei Jiao, Jixiang Li, Cailiang Fan, Liting Cao, Zuoyong Zhou, Shengping Wu, Chi Meng, Fengyuan Jiao, Gengxu Zhou
article en

Abstract

Zoonotic diseases not only cause great harm to animal health but also involve the development of animal husbandry, which in turn endangers human life and health and public health safety. Protein ubiquitination and autophagy are important ways for the body to degrade invading pathogens, which correspond to the ubiquitin (Ub)-proteasome system (UPS) and autophagic lysosomal pathway (ALP), respectively, and play an important role in the occurrence and development of diseases. For UPS, the substrate is delivered to the 26S proteasome system via a ubiquitination cascade and subsequently degraded and removed. For ALP, the substrate is encapsulated to form autophagosomes, which subsequently fuse with lysosomes to form autophagolysosomes, which are eventually degraded and cleared. However, a variety of zoonotic pathogens can interfere with the protein ubiquitination pathway and autophagy process to promote self-replication and survival, and resist host immune defense. This article reviews the mechanisms by which multiple pathogens interfere with protein degradation pathways, providing a new perspective for the treatment and prevention of zoonotic diseases.

Transboundary and Emerging DiseasesVol. 2025(1)
Southwest University (CN), Lanzhou Veterinary Research Institute (CN), China Animal Disease Control Center (CN), Chongqing Academy of Animal Science (CN)
Chinese Academy of Agricultural Sciences, Natural Science Foundation of Chongqing, State Key Laboratory of Veterinary Etiological Biology, Fundamental Research Funds for the Central Universities
Openalex Percentile: Top 94%
Toxoplasma gondii Research Studies
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