Baicalin Ameliorates L‐Glutamate‐Induced Hippocampal Oxidative Stress Injury and Apoptosis in Mice by Regulating Nrf2/HO‐1 Signalling Pathway

Baicalin can reduce the injury induced by L-glutamate, and the mechanism might be related to the activation of the Nrf2/HO-1 pathway.

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Publication Details

Journal
Basic & Clinical Pharmacology & Toxicology
Published
2025-03-20
DOI
https://doi.org/10.1111/bcpt.70024
Citations
1
Primary Topic
Biochemical effects in animals
Type
article
Field-Weighted Citation Impact
1.16

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article

Baicalin Ameliorates L‐Glutamate‐Induced Hippocampal Oxidative Stress Injury and Apoptosis in Mice by Regulating Nrf2/HO‐1 Signalling Pathway

Huiyang Gou, Jiarui Zheng, Mingjiang Yao, Feng Li et al.
1 citations
Basic & Clinical Pharmacology & Toxicology
Biochemical effects in animals
1.16
article

Baicalin Ameliorates L‐Glutamate‐Induced Hippocampal Oxidative Stress Injury and Apoptosis in Mice by Regulating Nrf2/HO‐1 Signalling Pathway

Huiyang Gou, Jiarui Zheng, Mingjiang Yao, Feng Li, Zishan Huang
article en
1 citations

Abstract

Baicalin can reduce the injury induced by L-glutamate, and the mechanism might be related to the activation of the Nrf2/HO-1 pathway.

Basic & Clinical Pharmacology & ToxicologyVol. 136(4)
Guangdong Pharmaceutical University (CN), Chinese Academy of Medical Sciences & Peking Union Medical College (CN), Xiyuan Hospital (CN)
Natural Science Foundation of Beijing Municipality, China Academy of Chinese Medical Sciences
Openalex Percentile: Top 24%
Biochemical effects in animals
1.16
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