Fc fragment of IgG binding protein suppresses tumor growth by stabilizing wild type P53 in colorectal cancer cells

The Fc fragment of IgG binding protein (FCGBP) exhibits differential expression across various tumor types. but its role in cancer progression remains underexplored. This research discovered that FCGBP is downregulated in colorectal cancer (CRC) cells and is negatively associated with poor prognosis. Overexpression of FCGBP inhibited the growth of P53 wild-type CRC cells both in vitro and in vivo. Mechanistically, immunoprecipitation experiments revealed that FCGBP competitively binds to MDM2, thereby attenuating the formation of the P53/MDM2 complex. This, in turn, reduces P53 ubiquitination and stabilizes the protein. Our findings reveal a novel mechanism through which FCGBP significantly inhibits CRC cell growth and propose a new targeted therapeutic strategy for CRC treatment.

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Publication Details

Journal
BMC Cancer
Published
2025-03-19
DOI
https://doi.org/10.1186/s12885-025-13873-y
Citations
4
Primary Topic
Cancer-related Molecular Pathways
Type
article
Field-Weighted Citation Impact
4.18
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article

Fc fragment of IgG binding protein suppresses tumor growth by stabilizing wild type P53 in colorectal cancer cells

Jiefu Wang, Xinran Lu, Yanpeng Yang, Junfeng Wang et al.
4 citations
BMC Cancer
Cancer-related Molecular Pathways
4.18
article

Fc fragment of IgG binding protein suppresses tumor growth by stabilizing wild type P53 in colorectal cancer cells

Jiefu Wang, Xinran Lu, Yanpeng Yang, Junfeng Wang, Jia Liu, Wenpeng Wang, Zhicheng Gong, Kai Liu
article en
4 citations

Abstract

The Fc fragment of IgG binding protein (FCGBP) exhibits differential expression across various tumor types. but its role in cancer progression remains underexplored. This research discovered that FCGBP is downregulated in colorectal cancer (CRC) cells and is negatively associated with poor prognosis. Overexpression of FCGBP inhibited the growth of P53 wild-type CRC cells both in vitro and in vivo. Mechanistically, immunoprecipitation experiments revealed that FCGBP competitively binds to MDM2, thereby attenuating the formation of the P53/MDM2 complex. This, in turn, reduces P53 ubiquitination and stabilizes the protein. Our findings reveal a novel mechanism through which FCGBP significantly inhibits CRC cell growth and propose a new targeted therapeutic strategy for CRC treatment.

BMC CancerVol. 25(1)
Peking University (CN), Tianjin Medical University Cancer Institute and Hospital (CN)
Good health and well-being
Openalex Percentile: Top 6%
Cancer-related Molecular Pathways
4.18
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Fc fragment of IgG binding protein suppresses tumor growth by stabilizing wild type P53 in colorectal cancer cells — Jiefu Wang, Xinran Lu, et al. · BMC Cancer (2025) | TGRS Research Map | TGRS