Protective effects of Apelin-13 on nicotine-induced H9c2 cardiomyocyte apoptosis and oxidative stress

Nicotine induction increases the oxidative stress and apoptotic capacity of H9c2 cardiomyocytes, but Apelin-13 protects H9c2 cardiomyocytes against nicotine-induced apoptosis and oxidative stress.

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Publication Details

Journal
Tobacco Induced Diseases
Published
2025-03-18
DOI
https://doi.org/10.18332/tid/201400
Citations
1
Primary Topic
Apelin-related biomedical research
Type
article
Field-Weighted Citation Impact
1.39
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article

Protective effects of Apelin-13 on nicotine-induced H9c2 cardiomyocyte apoptosis and oxidative stress

Can Xu, Luyang Zhou, Dongjin Wang, Ru Xu et al.
1 citations
Tobacco Induced Diseases
Apelin-related biomedical research
1.39
article

Protective effects of Apelin-13 on nicotine-induced H9c2 cardiomyocyte apoptosis and oxidative stress

Can Xu, Luyang Zhou, Dongjin Wang, Ru Xu, Xinyu Nie
article en
1 citations

Abstract

Nicotine induction increases the oxidative stress and apoptotic capacity of H9c2 cardiomyocytes, but Apelin-13 protects H9c2 cardiomyocytes against nicotine-induced apoptosis and oxidative stress.

Tobacco Induced DiseasesVol. 23(March)
Nanjing Medical University (CN), Nanjing University (CN)
Zero hunger
Openalex Percentile: Top 23%
Apelin-related biomedical research
1.39
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