EFNA2 Mediates Stiffness-Regulated Hypopharyngeal Cancer Progression
EFNA2 and elevated matrix stiffness jointly contribute to the malignant phenotype in HPC. EFNA2 may represent a potential therapeutic target for managing HPC progression induced by high matrix stiffness.
Authors
- Min Xu (ORCID: https://orcid.org/0000-0002-5587-5520)
- Bo You (ORCID: https://orcid.org/0000-0002-1065-2286)
- Yuanru Liu
- Tian Xia (ORCID: https://orcid.org/0000-0002-0097-6949)
- Songqun Hu (ORCID: https://orcid.org/0000-0002-7944-7985)
- Ying Shan (ORCID: https://orcid.org/0000-0001-8986-2325)
- Qicheng Zhang (ORCID: https://orcid.org/0000-0001-7277-0280)
- Huiting Liu
- Zhenxin Zhang (ORCID: https://orcid.org/0000-0003-1659-8951)
- Min Cheng (ORCID: https://orcid.org/0000-0003-2974-7418)
- Haosheng Ni
Institutions
- Nantong University (CN)
- Affiliated Hospital of Nantong University (CN)
- ZTT (China) (CN)
Publication Details
- Journal
- Clinical and Experimental Otorhinolaryngology
- Published
- 2025-03-17
- DOI
- https://doi.org/10.21053/ceo.2024.00315
- Primary Topic
- RNA modifications and cancer
- Type
- article
- Field-Weighted Citation Impact
- 0.00