Prenatal Stress Increases the Risk of the FPR2-related Dysfunction in the Brain's Resolution of Inflammation: A Study on the Humanized APP<sup>NL-F/NL-F</sup> Mouse Model of Alzheimer's Disease

Our results identify the FPR2 receptors as a driver regulating the RoI process in the brain and highlight that PS has diversified the picture of age-dependent neurodegenerative pathology.

Authors

J Frydrych
Agnieszka Basta‐Kaim (ORCID: https://orcid.org/0000-0002-3109-0040)
Ewa Trojan (ORCID: https://orcid.org/0000-0002-9573-7026)
Władysław Lasoń

Institutions

Maj Institute of Pharmacology (PL)
Polish Academy of Sciences (PL)

Publication Details

Journal: Current Neuropharmacology
Published: 2025-03-13
DOI: https://doi.org/10.2174/011570159x345385241004060055
Primary Topic: Tryptophan and brain disorders
Type: article
Field-Weighted Citation Impact: 0.00

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article

Prenatal Stress Increases the Risk of the FPR2-related Dysfunction in the Brain's Resolution of Inflammation: A Study on the Humanized APP^NL-F/NL-F Mouse Model of Alzheimer's Disease

J Frydrych, Agnieszka Basta‐Kaim, Ewa Trojan, Władysław Lasoń

Mar 13, 2025

Current Neuropharmacology

Tryptophan and brain disorders

article

Prenatal Stress Increases the Risk of the FPR2-related Dysfunction in the Brain's Resolution of Inflammation: A Study on the Humanized APP^NL-F/NL-F Mouse Model of Alzheimer's Disease

J Frydrych, Agnieszka Basta‐Kaim, Ewa Trojan, Władysław Lasoń

March 13, 2025article en

Abstract

Our results identify the FPR2 receptors as a driver regulating the RoI process in the brain and highlight that PS has diversified the picture of age-dependent neurodegenerative pathology.

Current NeuropharmacologyVol. 23(12)

Maj Institute of Pharmacology (PL), Polish Academy of Sciences (PL)

Openalex Percentile: Top 96%

Tryptophan and brain disorders

DOI Source OpenAlex

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Prenatal Stress Increases the Risk of the FPR2-related Dysfunction in the Brain's Resolution of Inflammation: A Study on the Humanized APP<sup>NL-F/NL-F</sup> Mouse Model of Alzheimer's Disease — J Frydrych, Agnieszka Basta‐Kaim, et al. · Current Neuropharmacology (2025) | TGRS Research Map | TGRS

Prenatal Stress Increases the Risk of the FPR2-related Dysfunction in the Brain's Resolution of Inflammation: A Study on the Humanized APP<sup>NL-F/NL-F</sup> Mouse Model of Alzheimer's Disease

Authors

Institutions

Publication Details

Prenatal Stress Increases the Risk of the FPR2-related Dysfunction in the Brain's Resolution of Inflammation: A Study on the Humanized APPNL-F/NL-F Mouse Model of Alzheimer's Disease

Prenatal Stress Increases the Risk of the FPR2-related Dysfunction in the Brain's Resolution of Inflammation: A Study on the Humanized APPNL-F/NL-F Mouse Model of Alzheimer's Disease

Abstract

Ask Laika to Summarize, Analyze, and Connect papers live on the map.

Prenatal Stress Increases the Risk of the FPR2-related Dysfunction in the Brain's Resolution of Inflammation: A Study on the Humanized APP^NL-F/NL-F Mouse Model of Alzheimer's Disease

Prenatal Stress Increases the Risk of the FPR2-related Dysfunction in the Brain's Resolution of Inflammation: A Study on the Humanized APP^NL-F/NL-F Mouse Model of Alzheimer's Disease