Physiological Modulation of Neutrophil Activation and Recruitment by Heat Shock Protein 90 in Acute Pancreatitis
Background and Objective: Severe acute pancreatitis (AP) is characterized by leukocyte infiltration and tissue necrosis, yet the cellular signaling pathways driving pancreatic damage remain poorly understood. Heat shock protein 90 (HSP90) is a key regulator of specific cellular functions, promoting pro-inflammatory responses. This study investigates the role of HSP90 signaling in acute pancreatitis.
Authors
- Zhian Sherzad Hayder
- Mohammed Merza (ORCID: https://orcid.org/0000-0002-3899-2231)
- Kewan Kamal Ahmad
- Roza Talaat Yaseen
- Huda Jabbar Jawhar
Institutions
- Hawler Medical University (IQ)
- Koya University (IQ)
- Tishk International University (IQ)
Publication Details
- Journal
- Cuestiones de Fisioterapia
- Published
- 2025-02-20
- DOI
- https://doi.org/10.48047/dwnp4j05
- Primary Topic
- Pancreatitis Pathology and Treatment
- Type
- article
- Field-Weighted Citation Impact
- 0.00