AMD1, a cardiotoxicity target for Maduramicin
Mad exerted cardiotoxic effects by upregulating the AMD1 gene, which may be associated with cell apoptosis, proliferation and inflammatory response. AMD1 also had cardiotoxicity function, by the impact of both myocardial cells and the microenvironment they live.
Authors
- Yuan Gao (ORCID: https://orcid.org/0000-0002-0670-9104)
- Xiangyu Pei (ORCID: https://orcid.org/0000-0003-1150-3001)
- J. Zheng
- Jie Zhao (ORCID: https://orcid.org/0000-0002-6450-5046)
- Ge Tian (ORCID: https://orcid.org/0009-0007-6170-2945)
- Ning Zhang (ORCID: https://orcid.org/0000-0002-1630-7686)
- Haiqing Liu (ORCID: https://orcid.org/0000-0001-6892-358X)
- Z. P. Xie (ORCID: https://orcid.org/0009-0006-1698-1060)
- Yuanlong Zhao (ORCID: https://orcid.org/0000-0002-9521-6191)
Institutions
- Jinzhou Medical University (CN)
Publication Details
- Journal
- BMC Pharmacology and Toxicology
- Published
- 2025-03-11
- DOI
- https://doi.org/10.1186/s40360-025-00897-0
- Primary Topic
- Coccidia and coccidiosis research
- Type
- article
- Field-Weighted Citation Impact
- 0.00