Tenascin-C Facilitates Microglial Polarization via TLR4/MyD88/NF-κB Pathway Following Subarachnoid Hemorrhage

In summary, the elevated level of Tenascin-C after subarachnoid hemorrhage induces the activation of microglia, releasing a large number of inflammatory factors and aggravating early brain injury.

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Publication Details

Journal
Journal of Inflammation Research
Published
2025-03-01
DOI
https://doi.org/10.2147/jir.s511378
Citations
4
Primary Topic
Immune cells in cancer
Type
article
Field-Weighted Citation Impact
3.19
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article

Tenascin-C Facilitates Microglial Polarization via TLR4/MyD88/NF-κB Pathway Following Subarachnoid Hemorrhage

Ruijie Ma, Jia-Qing Sun, Dayong Xia, Jiaqiang Liu et al.
4 citations
Journal of Inflammation Research
Immune cells in cancer
3.19
article

Tenascin-C Facilitates Microglial Polarization via TLR4/MyD88/NF-κB Pathway Following Subarachnoid Hemorrhage

Ruijie Ma, Jia-Qing Sun, Dayong Xia, Jiaqiang Liu, Niansheng Lai, Zhibin Hu, Jinlong Yuan, Min Peng
article en
4 citations

Abstract

In summary, the elevated level of Tenascin-C after subarachnoid hemorrhage induces the activation of microglia, releasing a large number of inflammatory factors and aggravating early brain injury.

Journal of Inflammation ResearchVol. Volume 18
Wannan Medical College (CN), Xuzhou Medical College (CN), Nanjing Drum Tower Hospital (CN), First Affiliated Hospital of Wannan Medical College (CN)
Good health and well-being
Openalex Percentile: Top 9%
Immune cells in cancer
3.19
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