SNX10 functions as a modulator of piecemeal mitophagy and mitochondrial bioenergetics

We here identify the endosomal protein SNX10 as a negative regulator of piecemeal mitophagy of OXPHOS machinery components. In control conditions, SNX10 localizes to early endocytic compartments in a PtdIns3P-dependent manner and modulates endosomal trafficking but also shows dynamic connections with mitochondria. Upon hypoxia-mimicking conditions, SNX10 localizes to late endosomal structures containing selected mitochondrial proteins, including COX-IV and SAMM50, and the autophagy proteins SQSTM1/p62 and LC3B. The turnover of COX-IV was enhanced in SNX10-depleted cells, with a corresponding reduced mitochondrial respiration and citrate synthase activity. Importantly, zebrafish larvae lacking Snx10 show reduced levels of Cox-IV, as well as elevated ROS levels and ROS-mediated cell death in the brain, demonstrating the in vivo relevance of SNX10-mediated modulation of mitochondrial bioenergetics.

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Publication Details

Journal
The Journal of Cell Biology
Published
2025-02-05
DOI
https://doi.org/10.1083/jcb.202404009
Citations
4
Primary Topic
Autophagy in Disease and Therapy
Type
article
Field-Weighted Citation Impact
5.42

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article

SNX10 functions as a modulator of piecemeal mitophagy and mitochondrial bioenergetics

Benan John Mathai, Sebastian W. Schultz, Nagham Asp, Sakshi Singh et al.
4 citations
The Journal of Cell Biology
Autophagy in Disease and Therapy
5.42
article

SNX10 functions as a modulator of piecemeal mitophagy and mitochondrial bioenergetics

Benan John Mathai, Sebastian W. Schultz, Nagham Asp, Sakshi Singh, Serhiy Pankiv, Anne Simonsen, Ana Lapão, Laura Trachsel-Moncho, Chiara Veroni
article en
4 citations

Abstract

We here identify the endosomal protein SNX10 as a negative regulator of piecemeal mitophagy of OXPHOS machinery components. In control conditions, SNX10 localizes to early endocytic compartments in a PtdIns3P-dependent manner and modulates endosomal trafficking but also shows dynamic connections with mitochondria. Upon hypoxia-mimicking conditions, SNX10 localizes to late endosomal structures containing selected mitochondrial proteins, including COX-IV and SAMM50, and the autophagy proteins SQSTM1/p62 and LC3B. The turnover of COX-IV was enhanced in SNX10-depleted cells, with a corresponding reduced mitochondrial respiration and citrate synthase activity. Importantly, zebrafish larvae lacking Snx10 show reduced levels of Cox-IV, as well as elevated ROS levels and ROS-mediated cell death in the brain, demonstrating the in vivo relevance of SNX10-mediated modulation of mitochondrial bioenergetics.

The Journal of Cell BiologyVol. 224(5)
Oslo University Hospital (NO), University of Oslo (NO)
Kreftforeningen, Universitetet i Oslo, Norges Forskningsråd, Helse Sør-Øst RHF, H2020 Marie Skłodowska-Curie Actions
Life below water
Openalex Percentile: Top 5%
Autophagy in Disease and Therapy
5.42
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SNX10 functions as a modulator of piecemeal mitophagy and mitochondrial bioenergetics — Benan John Mathai, Sebastian W. Schultz, et al. · The Journal of Cell Biology (2025) | TGRS Research Map | TGRS