Cxcl9-deficiency attenuates the progression of post-traumatic osteoarthritis in mice
Our results point towards a pro-inflammatory role of CXCL9 in OA and identify a potential new target for the pharmacological treatment of OA.
Authors
- Shan Jiang (ORCID: https://orcid.org/0000-0002-1545-2152)
- Johannes Keller (ORCID: https://orcid.org/0000-0002-8925-7288)
- Laura Brylka (ORCID: https://orcid.org/0000-0003-4734-9966)
- Anke Baranowsky (ORCID: https://orcid.org/0009-0007-3883-646X)
- Antonia Donat (ORCID: https://orcid.org/0000-0003-3069-0199)
- Tim Rolvien (ORCID: https://orcid.org/0000-0003-1058-1307)
- Karl‐Heinz Frosch (ORCID: https://orcid.org/0000-0002-9195-1869)
- Weixin Xie (ORCID: https://orcid.org/0000-0002-1404-793X)
- Thorsten Schinke
Institutions
- Universität Hamburg (DE)
- University Medical Center Hamburg-Eppendorf (DE)
- BG Klinikum Hamburg (DE)
Publication Details
- Journal
- Inflammation Research
- Published
- 2025-03-06
- DOI
- https://doi.org/10.1007/s00011-025-02013-8
- Citations
- 1
- Primary Topic
- Osteoarthritis Treatment and Mechanisms
- Type
- article
- Field-Weighted Citation Impact
- 1.67