Anti-Leukotriene Receptor Blockers Improve Tendon-Bone Interface Healing in a Rat Model of Acute Rotator Cuff Tear

The use of MS improved tendon elasticity through suppressing fatty infiltration and improving TBI collagen density and arrangement. The mechanism is down-regulation of IL-1β and IL-6. These results strongly support the use of MS as an anti-inflammatory agent that does not impair tendon healing. [<i>Orthopedics.</i> 2025;48(2):e105-e112.].

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Publication Details

Journal
Orthopedics
Published
2025-03-01
DOI
https://doi.org/10.3928/01477447-20250218-01
Citations
2
Primary Topic
Shoulder Injury and Treatment
Type
article
Field-Weighted Citation Impact
4.01
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article

Anti-Leukotriene Receptor Blockers Improve Tendon-Bone Interface Healing in a Rat Model of Acute Rotator Cuff Tear

Sungjin Park, Seok Won Chung, Hyun‐Joo Lee, Dong‐Hyun Kim et al.
2 citations
Orthopedics
Shoulder Injury and Treatment
4.01
article

Anti-Leukotriene Receptor Blockers Improve Tendon-Bone Interface Healing in a Rat Model of Acute Rotator Cuff Tear

Sungjin Park, Seok Won Chung, Hyun‐Joo Lee, Dong‐Hyun Kim, Yun Choi, Jun-Young Kim, Jong Pil Yoon
article en
2 citations

Abstract

The use of MS improved tendon elasticity through suppressing fatty infiltration and improving TBI collagen density and arrangement. The mechanism is down-regulation of IL-1β and IL-6. These results strongly support the use of MS as an anti-inflammatory agent that does not impair tendon healing. [<i>Orthopedics.</i> 2025;48(2):e105-e112.].

OrthopedicsVol. 48(2)
Kyungpook National University (KR)
Openalex Percentile: Top 8%
Shoulder Injury and Treatment
4.01
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