Aspirin impedes non-small cell lung cancer development via fine-tuning the CD36 localization regulated by GPIHBP1
Our findings provide new insights into the mechanism by which aspirin suppresses lung cancer development in a GPIHBP1-dependent manner and may provide a promising target in NSCLC treatment.
Authors
- Katsuhiro Okuda (ORCID: https://orcid.org/0000-0003-0099-2348)
- Jia Chen (ORCID: https://orcid.org/0000-0003-0301-4494)
- Wei Liu (ORCID: https://orcid.org/0000-0001-5568-2975)
- Ya Gao (ORCID: https://orcid.org/0000-0003-2244-7834)
- Dan Qiao (ORCID: https://orcid.org/0000-0003-2375-1512)
- Yoshihisa Shimada (ORCID: https://orcid.org/0000-0003-3811-6159)
- Linong Yao
Institutions
- Tokyo Medical University (JP)
- Nagoya City University (JP)
- Northwest University (CN)
Publication Details
- Journal
- Translational Lung Cancer Research
- Published
- 2025-02-01
- DOI
- https://doi.org/10.21037/tlcr-2024-1174
- Citations
- 1
- Primary Topic
- Peptidase Inhibition and Analysis
- Type
- article
- Field-Weighted Citation Impact
- 1.05