Inflammation-Induced Nitric Oxide Synthase May Mediate the Acute Hypotensive Effect of ST36 Stimulation
These results indicated that MA at ST36 induced inflammation, which activated NOS and resulted in the release of nitric oxide. This led to the vasodilation of peripheral vessels and transient hypotension.
Authors
- Xin Cao (ORCID: https://orcid.org/0000-0002-3992-3377)
- Li‐Juan Zhu (ORCID: https://orcid.org/0000-0003-3478-1725)
- Qian Lei (ORCID: https://orcid.org/0000-0001-8647-1011)
- Xinghuai Sun (ORCID: https://orcid.org/0000-0002-4440-1389)
- Yue Xing (ORCID: https://orcid.org/0009-0004-3716-8621)
- Hong Hao (ORCID: https://orcid.org/0000-0002-4749-1834)
- Zili Tang
- Shuqing Liu
Institutions
- Soochow University (CN)
- First Affiliated Hospital of Soochow University (CN)
- Chengdu University of Traditional Chinese Medicine (CN)
Publication Details
- Journal
- Journal of Inflammation Research
- Published
- 2025-02-01
- DOI
- https://doi.org/10.2147/jir.s494037
- Primary Topic
- Nitric Oxide and Endothelin Effects
- Type
- article
- Field-Weighted Citation Impact
- 0.00
Funders
- National Natural Science Foundation of China
- Natural Science Foundation of Sichuan Province