GluOC Induced SLC7A11 and SLC38A1 to Activate Redox Processes and Resist Ferroptosis in TNBC

This study elucidated the mechanism of GluOC in inhibiting ferroptosis in TNBC. The findings not only provided new insights into ferroptosis but also potentially offered new concepts for the development of future anticancer therapies, which may contribute to improving the treatment of TNBC patients.

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Publication Details

Journal
Cancers
Published
2025-02-21
DOI
https://doi.org/10.3390/cancers17050739
Citations
1
Primary Topic
Ferroptosis and cancer prognosis
Type
article
Field-Weighted Citation Impact
1.52

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article

GluOC Induced SLC7A11 and SLC38A1 to Activate Redox Processes and Resist Ferroptosis in TNBC

Ping Ma, Jiaojiao Xu, Xue Bai, Qian Du et al.
1 citations
Cancers
Ferroptosis and cancer prognosis
1.52
article

GluOC Induced SLC7A11 and SLC38A1 to Activate Redox Processes and Resist Ferroptosis in TNBC

Ping Ma, Jiaojiao Xu, Xue Bai, Qian Du, Ziqian Zhang, Jianhong Yang, Keting Dong
article en
1 citations

Abstract

This study elucidated the mechanism of GluOC in inhibiting ferroptosis in TNBC. The findings not only provided new insights into ferroptosis but also potentially offered new concepts for the development of future anticancer therapies, which may contribute to improving the treatment of TNBC patients.

CancersVol. 17(5)
University of Chinese Academy of Sciences (CN)
Fundamental Research Funds for the Central Universities
Good health and well-being
Openalex Percentile: Top 22%
Ferroptosis and cancer prognosis
1.52
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