Super-resolution of nodal and paranodal disruption in anti-pan-neurofascin-associated autoimmune nodopathy
These likely reversible changes may explain the rapid recovery seen in patients with anti-pan-neurofascin autoantibodies following autoantibody depletion. Conversely, the small percentage of severely and axonally damaged nodes may account for the residual symptoms experienced by most patients.
Authors
- Claudia Sommer (ORCID: https://orcid.org/0000-0002-7064-5002)
- Luise Appeltshauser (ORCID: https://orcid.org/0000-0001-5248-4009)
- Dirk Brämer
- Thomas-Otavio Peulen (ORCID: https://orcid.org/0000-0001-8478-9755)
- Katrin G. Heinze (ORCID: https://orcid.org/0000-0003-2372-6800)
- Kathrin Doppler (ORCID: https://orcid.org/0000-0003-2883-0009)
- Janis Linke (ORCID: https://orcid.org/0000-0002-4943-4542)
- Christian Geis (ORCID: https://orcid.org/0000-0002-9859-581X)
- Vinicius da Cruz Neris Geßner
Institutions
- University of Würzburg (DE)
- Universitätsklinikum Würzburg (DE)
- Jena University Hospital (DE)
Publication Details
- Journal
- Frontiers in Immunology
- Published
- 2025-02-20
- DOI
- https://doi.org/10.3389/fimmu.2025.1540859
- Citations
- 2
- Primary Topic
- Peripheral Neuropathies and Disorders
- Type
- article
- Field-Weighted Citation Impact
- 2.78
Funders
- Deutsche Forschungsgemeinschaft