GPR120/FFAR4 protects retinal vascular endothelial cells against high glucose injury via suppressing ROS-ERS mediated apoptosis

GPR120 protects HRVECs against HG induced apoptosis, and suppressing ROS-ERS pathway is one of the mechanisms involved. Activation of GPR120 may be considered as a potential therapeutic target for diabetic retinopathy.

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Publication Details

Journal
International Journal of Ophthalmology
Published
2025-02-20
DOI
https://doi.org/10.18240/ijo.2025.03.02
Citations
1
Primary Topic
Retinal Diseases and Treatments
Type
article
Field-Weighted Citation Impact
1.71
Controls
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article

GPR120/FFAR4 protects retinal vascular endothelial cells against high glucose injury via suppressing ROS-ERS mediated apoptosis

Li-Zhao Wang
1 citations
International Journal of Ophthalmology
Retinal Diseases and Treatments
1.71
article

GPR120/FFAR4 protects retinal vascular endothelial cells against high glucose injury via suppressing ROS-ERS mediated apoptosis

Li-Zhao Wang
article en
1 citations

Abstract

GPR120 protects HRVECs against HG induced apoptosis, and suppressing ROS-ERS pathway is one of the mechanisms involved. Activation of GPR120 may be considered as a potential therapeutic target for diabetic retinopathy.

International Journal of OphthalmologyVol. 18(3)
China XD Group (China) (CN)
Openalex Percentile: Top 22%
Retinal Diseases and Treatments
1.71
DOIOpen Access PDFOpenAlex
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GPR120/FFAR4 protects retinal vascular endothelial cells against high glucose injury via suppressing ROS-ERS mediated apoptosis — Li-Zhao Wang · International Journal of Ophthalmology (2025) | TGRS Research Map | TGRS