<i>Ntoco</i> Promotes Ferroptosis via Hnrnpab‐Mediated <scp>NF</scp>‐<scp>κB</scp>/Lcn2 Axis Following Traumatic Brain Injury in Mice
Our findings suggest that Ntoco plays a crucial role in TBI-induced ferroptosis by modulating the NF-κB/Lcn2 signaling pathway. Targeting Ntoco may provide a promising therapeutic strategy to mitigate ferroptosis and improve outcomes following TBI.
Authors
- Linbo Gao (ORCID: https://orcid.org/0000-0002-7572-8990)
- Huaqin Sun (ORCID: https://orcid.org/0000-0002-4548-4657)
- Hanjun Zuo
- Qiang Wang (ORCID: https://orcid.org/0000-0002-6141-0068)
- Zhao Wang (ORCID: https://orcid.org/0000-0003-4897-9986)
- Xiao Xiao
- Tao Wang (ORCID: https://orcid.org/0000-0002-8955-416X)
- Tingyu Li (ORCID: https://orcid.org/0000-0002-1940-7076)
- Juanjuan Li (ORCID: https://orcid.org/0000-0003-2164-094X)
- Yanyun Wang (ORCID: https://orcid.org/0000-0002-8102-8851)
- Xiaolei Luo
Institutions
- Sichuan University (CN)
- Kunming Medical University (CN)
- University of Hong Kong (HK)
Publication Details
- Journal
- CNS Neuroscience & Therapeutics
- Published
- 2025-02-01
- DOI
- https://doi.org/10.1111/cns.70282
- Citations
- 2
- Primary Topic
- Cancer-related molecular mechanisms research
- Type
- article
- Field-Weighted Citation Impact
- 1.87
Funders
- National Natural Science Foundation of China
- Huazhong University of Science and Technology