Study on the Molecular Mechanism of Baicalin Phosphorylation of Tau Protein Content in a Cell Model of Intervention Cognitive Impairment

As demonstrated by the present study, the low concentration of baicalein (15μmol/L) effectively managed the mild cognitive impairment by regulating the phosphorylation of AKT1 and GSK-3β.

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Publication Details

Journal
Neuropsychiatric Disease and Treatment
Published
2025-02-01
DOI
https://doi.org/10.2147/ndt.s482362
Citations
1
Primary Topic
Neurological Disease Mechanisms and Treatments
Type
article
Field-Weighted Citation Impact
0.84
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article

Study on the Molecular Mechanism of Baicalin Phosphorylation of Tau Protein Content in a Cell Model of Intervention Cognitive Impairment

Dong-Hui Guan, Canbin Zhao, Chunmei Liu, Juncheng Li
1 citations
Neuropsychiatric Disease and Treatment
Neurological Disease Mechanisms and Treatments
0.84
article

Study on the Molecular Mechanism of Baicalin Phosphorylation of Tau Protein Content in a Cell Model of Intervention Cognitive Impairment

Dong-Hui Guan, Canbin Zhao, Chunmei Liu, Juncheng Li
article en
1 citations

Abstract

As demonstrated by the present study, the low concentration of baicalein (15μmol/L) effectively managed the mild cognitive impairment by regulating the phosphorylation of AKT1 and GSK-3β.

Neuropsychiatric Disease and TreatmentVol. Volume 21
Shandong University of Traditional Chinese Medicine (CN), Affiliated Hospital of Shandong University of Traditional Chinese Medicine (CN)
Openalex Percentile: Top 30%
Neurological Disease Mechanisms and Treatments
0.84
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