In Vitro Inhibition of Endoplasmic Reticulum Stress: A Promising Therapeutic Strategy for Patients with Crohn’s Disease

ER stress and UPR pathways are activated in CD colonic mucosa, and PBA reduces these markers, suggesting potential therapeutic benefits for CD-related inflammation.

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Publication Details

Journal
Cells
Published
2025-02-13
DOI
https://doi.org/10.3390/cells14040270
Citations
4
Primary Topic
Galectins and Cancer Biology
Type
article
Field-Weighted Citation Impact
3.19

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article

In Vitro Inhibition of Endoplasmic Reticulum Stress: A Promising Therapeutic Strategy for Patients with Crohn’s Disease

Lívia Bitencourt Pascoal, Lı́cio A. Velloso, Lívia Moreira Genaro, Priscilla de Sene Portel Oliveira et al.
4 citations
Cells
Galectins and Cancer Biology
3.19
article

In Vitro Inhibition of Endoplasmic Reticulum Stress: A Promising Therapeutic Strategy for Patients with Crohn’s Disease

Lívia Bitencourt Pascoal, Lı́cio A. Velloso, Lívia Moreira Genaro, Priscilla de Sene Portel Oliveira, Maria de Lourdes Setsuko Ayrizono, Michel Gardere Camargo, Beatriz Alves Guerra, Raquel Franco Leal, Andressa Coope, Bruno Lima Rodrigues, Leonardo Saint Clair Assad Warrak
article en
4 citations

Abstract

ER stress and UPR pathways are activated in CD colonic mucosa, and PBA reduces these markers, suggesting potential therapeutic benefits for CD-related inflammation.

CellsVol. 14(4)
Universidade Estadual de Campinas (UNICAMP) (BR)
Fundação de Amparo à Pesquisa do Estado de São Paulo, Conselho Nacional de Desenvolvimento Científico e Tecnológico
Openalex Percentile: Top 9%
Galectins and Cancer Biology
3.19
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