Inhibition of complement system-related gene ITGB2 attenuates epithelial–mesenchymal transition and inflammation in diabetic nephropathy

Complement system-related gene ITGB2 was overexpressed in DN, and inhibition of ITGB2 attenuated EMT and inflammation in DN.

Authors

Institutions

Publication Details

Journal
European journal of medical research
Published
2025-02-08
DOI
https://doi.org/10.1186/s40001-025-02323-x
Citations
2
Primary Topic
Chronic Kidney Disease and Diabetes
Type
article
Field-Weighted Citation Impact
2.86
Controls
|||
ALL TIME
JAN
FEB
MAR
APR
MAY
JUN
JUL
AUG
SEP
OCT
article

Inhibition of complement system-related gene ITGB2 attenuates epithelial–mesenchymal transition and inflammation in diabetic nephropathy

Jun Peng, Lu Zhou, Wenqi Zhao, Kun Ding
2 citations
European journal of medical research
Chronic Kidney Disease and Diabetes
2.86
article

Inhibition of complement system-related gene ITGB2 attenuates epithelial–mesenchymal transition and inflammation in diabetic nephropathy

Jun Peng, Lu Zhou, Wenqi Zhao, Kun Ding
article en
2 citations

Abstract

Complement system-related gene ITGB2 was overexpressed in DN, and inhibition of ITGB2 attenuated EMT and inflammation in DN.

European journal of medical researchVol. 30(1)
Central Hospital of Wuhan (CN), Chinese People's Liberation Army (CN)
Zero hunger
Openalex Percentile: Top 12%
Chronic Kidney Disease and Diabetes
2.86
AI Navigator

Ask Laika to Summarize, Analyze, and Connect papers live on the map.

Summarize Papers & Methodologies

Extract key findings, datasets, and comparative methods across publications.

Benchmark Rankings & Visual Analytics

Rank top research institutions, authors, funders, topics, and journals by Field-Weighted Citation Impact (FWCI) and paper volume with instant charts.

Connect Distant Disciplines

Bridge topological clusters on the map to find hidden collaborative intersections.