MiR-223 within neutrophil axis promotes Th17 expansion by PI3K-AKT pathway in systemic lupus erythematosus

MiR-223 within neutrophil axis contributes to Th17 expansion by PI3K-AKT pathway in SLE, and miR-223 could be a therapeutic target of SLE.

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Publication Details

Journal
Arthritis Research & Therapy
Published
2025-02-03
DOI
https://doi.org/10.1186/s13075-025-03487-x
Citations
2
Primary Topic
Cytokine Signaling Pathways and Interactions
Type
article
Field-Weighted Citation Impact
2.09

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article

MiR-223 within neutrophil axis promotes Th17 expansion by PI3K-AKT pathway in systemic lupus erythematosus

Yan Lu, Cheng‐Zhong Zhang
2 citations
Arthritis Research & Therapy
Cytokine Signaling Pathways and Interactions
2.09
article

MiR-223 within neutrophil axis promotes Th17 expansion by PI3K-AKT pathway in systemic lupus erythematosus

Yan Lu, Cheng‐Zhong Zhang
article en
2 citations

Abstract

MiR-223 within neutrophil axis contributes to Th17 expansion by PI3K-AKT pathway in SLE, and miR-223 could be a therapeutic target of SLE.

Arthritis Research & TherapyVol. 27(1)
Nanjing Medical University (CN)
National Natural Science Foundation of China
Openalex Percentile: Top 14%
Cytokine Signaling Pathways and Interactions
2.09
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MiR-223 within neutrophil axis promotes Th17 expansion by PI3K-AKT pathway in systemic lupus erythematosus — Yan Lu, Cheng‐Zhong Zhang · Arthritis Research & Therapy (2025) | TGRS Research Map | TGRS