Inactivation of Notch1-TGF-β-Smads Signaling Pathway by Atorvastatin Improves Cardiac Function and Hemodynamic Performance in Acute Myocardial Infarction Rats

Atorvastatin inhibited myocardial fibrosis by interfering with notch1-TGF-β-smads signal pathways. In addition, it could mitigate myocardial remodeling and improve cardiac functions and hemodynamics.

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Publication Details

Journal
Journal of Vascular Research
Published
2025-01-27
DOI
https://doi.org/10.1159/000542728
Citations
3
Primary Topic
Cardiac Fibrosis and Remodeling
Type
article
Field-Weighted Citation Impact
4.16
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article

Inactivation of Notch1-TGF-β-Smads Signaling Pathway by Atorvastatin Improves Cardiac Function and Hemodynamic Performance in Acute Myocardial Infarction Rats

Taniya Fernando, Mengyun Yang, Qi Kang, Mei Ling Kang
3 citations
Journal of Vascular Research
Cardiac Fibrosis and Remodeling
4.16
article

Inactivation of Notch1-TGF-β-Smads Signaling Pathway by Atorvastatin Improves Cardiac Function and Hemodynamic Performance in Acute Myocardial Infarction Rats

Taniya Fernando, Mengyun Yang, Qi Kang, Mei Ling Kang
article en
3 citations

Abstract

Atorvastatin inhibited myocardial fibrosis by interfering with notch1-TGF-β-smads signal pathways. In addition, it could mitigate myocardial remodeling and improve cardiac functions and hemodynamics.

Journal of Vascular ResearchVol. 62(3)
Tianjin Medical University General Hospital (CN)
Good health and well-being
Openalex Percentile: Top 7%
Cardiac Fibrosis and Remodeling
4.16
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