WCN25-2391 Acid-Bace Balance of Renal Venous Blood in Patients with Secondary Hypertension

dyscrasias (3.9 +/-1.9 vs 2.8+/-0.4mg/dl, p0.03).We hypothesize that this is probably due to Fanconi's syndrome secondary to plasma cell dyscrasia.While two deaths were related to malignancy, one succumbed to calciphylaxis.Treatment strategies in patients without CKD included furosemide with IV fluids (88%), salmon calcitonin (35%), bisphosphonate (30%), hemodialysis (17%) and cinacalcet (12%).All patients with PTH adenoma were successfully treated with resection surgery.Majority of hypercalcemia in CKD patients was due to unmonitored consumption of calcium based phosphate binders and vitamin D supplements.Treatment in these patients mainly involved usage of non-calcium based phosphate binders, cinacalcet and low calcium bath hemodialysis.However, two patients with CKD were detected to have tuberculosis and hypercalcemia resolved after anti-tubercular treatment.Conclusions: Lower phosphate levels, high PTH levels, suppressed PTH and high alkaline phosphatase levels in hypercalcemia should raise suspicion of plasma cell dyscrasias, non myelomatous malignancies, iatrogenic causes and malignancies respectively.The use of calcium based phosphate binders and vitamin D supplements in CKD should be appropriately monitored.Tuberculosis in developing countries is a readily treatable cause of hypercalcemia.I have no potential conflict of interest to disclose.I did not use generative AI and AI-assisted technologies in the writing process.